Ac-SDKP attenuates ER stress-stimulated collagen production in cardiac fibroblasts by inhibiting CHOP-mediated NF-κB expression

Inflammation and cardiac fibrosis are prevalent pathophysiologic conditions associated with hypertension, cardiac remodeling, and heart failure. Endoplasmic reticulum (ER) stress triggers the cells to activate unfolded protein responses (UPRs) and upregulate the ER stress chaperon, enzymes, and down...

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Autors principals:	Hamid Suhail, Hongmei Peng, Khalid Matrougui, Nour-Eddine Rhaleb
Format:	Artigo
Idioma:	Inglês
Publicat:	Frontiers Media S.A. 2024-03-01
Col·lecció:	Frontiers in Pharmacology
Matèries:	Ac-SDKP ER stress UPRs inflammation collagen human cardiac fibroblast
Accés en línia:	https://www.frontiersin.org/articles/10.3389/fphar.2024.1352222/full
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