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Zmynd11 is essential for neurogenesis by coordinating H3K36me3 modification of Epha2 and PI3K signaling pathway

Abstract 10p15.3 deletion syndrome is caused by the deficiency of MYND-type zinc finger domain-containing protein 11 (ZMYND11) and featured by global developmental delay, intellectual disability, behavioral abnormalities, etc. Although the roles of Zmynd11 is intensively studied in cancer, the funct...

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Hlavní autoři: Xu Yang, Lan Li, Wenzheng Qu, Xuejun Cheng, Jinyu Zhang, Yan Sun, Suxiao Liu, Guoping Peng, Rui Zheng, Xuekun Li
Médium: Artigo
Jazyk:Inglês
Vydáno: BMC 2025-04-01
Edice:Cell & Bioscience
Témata:
10p15.3 deletion syndrome
Zmynd11
Neurogenesis
H3K36me3 modification
Epha2
PI3K
On-line přístup:https://doi.org/10.1186/s13578-025-01392-z
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