4,8-Dicarboxyl-8,9-Iridoid-1-Glycoside Alleviates Cardiac Dysfunction After Myocardial Ischaemia-Reperfusion Injury by Activating the PI3K/AKT Pathway

Background Extracellular matrix (ECM) deposition and excessive fibrosis are important factors in the deterioration of cardiac function after myocardial ischaemia‒reperfusion injury (I/RI). However, therapeutic strategies for inhibiting ECM deposition and excessive fibrosis have still not been elucid...

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Hlavní autoři:	AiJiao Sun, CuiYu Yu, FeiYan Zhu, Jing Chen, LiangChuan Chen, ZePing Hu MD, WeiBing Wang MD
Médium:	Artigo
Jazyk:	Inglês
Vydáno:	SAGE Publishing 2026-04-01
Edice:	Dose-Response
On-line přístup:	https://doi.org/10.1177/15593258261444840
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