Dihydromyricetin suppresses endothelial NLRP3 inflammasome activation and attenuates atherogenesis by promoting mitophagy

Abstract Background NOD-like receptor protein 3 (NLRP3) inflammasome activation is indispensable for atherogenesis. Mitophagy has emerged as a potential strategy to counteract NLRP3 inflammasome activation triggered by impaired mitochondria. Our previous research has indicated that dihydromyricetin,...

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Egile Nagusiak:	Qin Hu, Chengying Li, Ting Zhang, Long Yi, Yifan Shan, Xiangyu Ma, Tongjian Cai, Li Ran, Hui Shen, Yafei Li
Formatua:	Artigo
Hizkuntza:	Inglês
Argitaratua:	BMC 2024-09-01
Saila:	Lipids in Health and Disease
Gaiak:	Atherosclerosis Dihydromyricetin Mitophagy NLRP3 inflammasome Vascular endothelium
Sarrera elektronikoa:	https://doi.org/10.1186/s12944-024-02263-1
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