Eleutheroside E Attenuates Doxorubicin-Induced Cardiotoxicity by Suppressing Ferroptosis Through Activation of the Nrf2/SLC7A11/GPX4 Signaling Pathway

Doxorubicin (DOX)-induced cardiotoxicity significantly impairs cancer patient survival rates. Eleutheroside E (EE), a polyphenolic compound with established cardioprotective properties against high-altitude myocardial injury and ischemia/reperfusion damage, has not previously been investigated in th...

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Detaylı Bibliyografya

Asıl Yazarlar:	Peng Sun MD, Liheng Chen MD, Xiangzhou Chen MD, Xuwei Zhang MD, Junjie Guan MD, Hongwei Mo MD, Yu Liang MD, Jingchao Li MD, Jing Yan MD, Deshu Chen MD, Chongbin Zhong MD, Pingzhen Yang MD
Materyal Türü:	Artigo
Dil:	Inglês
Baskı/Yayın Bilgisi:	SAGE Publishing 2026-03-01
Seri Bilgileri:	Journal of Cardiovascular Pharmacology and Therapeutics
Online Erişim:	https://doi.org/10.1177/10742484261428559
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