CCL5 via GPX1 activation protects hippocampal memory function after mild traumatic brain injury

Traumatic brain injury (TBI) is a prevalent head injury worldwide which increases the risk of neurodegenerative diseases. Increased reactive oxygen species (ROS) and inflammatory chemokines after TBI induces secondary effects which damage neurons. Targeting NADPH oxidase or increasing redox systems...

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Detalhes bibliográficos

Principais autores:	Man-Hau Ho, Chia-Hung Yen, Tsung-Hsun Hsieh, Tzu-Jen Kao, Jing-Yuan Chiu, Yung-Hsiao Chiang, Barry J. Hoffer, Wen-Chang Chang, Szu-Yi Chou
Formato:	Artigo
Idioma:	Inglês
Publicado em:	Elsevier 2021-10-01
Colecção:	Redox Biology
Assuntos:	CCL5 Reactive oxygen species (ROS) NADPH oxidase Traumatic brain injury Glutathione peroxidase - 1 (GPX1)
Acesso em linha:	http://www.sciencedirect.com/science/article/pii/S2213231721002263
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