Ibrutinib Contributes to Atrial Arrhythmia through the Autophagic Degradation of Connexins by Inhibiting the PI3K-AKT-mTOR Signaling Pathway
Background: Ibrutinib could increase the risk of atrial fibrillation (AF) in chronic lymphocytic leukemia (CLL) patients. However, the precise mechanism underlying ibrutinib-induced AF remains incompletely elucidated. Methods: We investigated the proportion of ibrutinib-treated CLL patients with new...
محفوظ في:
| المؤلفون الرئيسيون: | , , , , , , , , |
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| التنسيق: | Artigo |
| اللغة: | Inglês |
| منشور في: |
IMR Press
2024-05-01
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| سلاسل: | Frontiers in Bioscience-Landmark |
| الموضوعات: | |
| الوصول للمادة أونلاين: | https://www.imrpress.com/journal/FBL/29/5/10.31083/j.fbl2905201 |
| الوسوم: |
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