Lack of Contribution of p66shc and Its Mitochondrial Translocation to Ischemia-Reperfusion Injury and Cardioprotection by Ischemic Preconditioning

Whereas high amounts of reactive oxygen species (ROS) contribute to cardiac damage following ischemia and reperfusion (IR), low amounts function as trigger molecules in the cardioprotection by ischemic preconditioning (IPC). The mitochondrial translocation and contribution of the hydrogen peroxide-g...

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Auteurs principaux:	Kerstin Boengler, Péter Bencsik, János Palóczi, Krisztina Kiss, Márton Pipicz, Judit Pipis, Péter Ferdinandy, Klaus-Dieter Schlüter, Rainer Schulz
Format:	Artigo
Langue:	Inglês
Publié:	Frontiers Media S.A. 2017-10-01
Collection:	Frontiers in Physiology
Sujets:	ischemia/reperfusion ischemic preconditioning reactive oxygen species mitochondria p66shc
Accès en ligne:	http://journal.frontiersin.org/article/10.3389/fphys.2017.00733/full
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