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CXCL9 and CXCL10 support the exacerbated humoral response in recovered COVID-19 patients who developed acute respiratory distress syndrome by promoting plasma cell differentiation, whereas CXCL9 also induces CD40L and CXCR3 upregulation on T helper cells

BackgroundSevere COVID-19 is frequently associated with acute respiratory distress syndrome (ARDS) and prolonged pulmonary sequelae. Persistent immune activation, including dysregulated B cell responses and increased proinflammatory chemokines, has been linked to the post-acute sequelae of SARS-CoV-...

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Principais autores: Romina Quiroga, Sergio Sanhueza, Catalina Sepúlveda, Bárbara Antilef, Camila Muñoz, Camilo Cabrera, Marco Fraga, Faryd Llerena, Liliana Lamperti, María Inés Barría, Alicia Colombo, Gonzalo Labarca, Mario Henríquez-Beltrán, Luciano Ferrada, Estefanía Nova-Lamperti
Formato: Artigo
Idioma:Inglês
Publicado em: Frontiers Media S.A. 2025-12-01
coleção:Frontiers in Immunology
Assuntos:
COVID-19
IgG
antibodies
B cells
plasmablasts
chemokines
Acesso em linha:https://www.frontiersin.org/articles/10.3389/fimmu.2025.1684704/full
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