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A potential therapeutic effect of catalpol in Duchenne muscular dystrophy revealed by binding with TAK1

Background Duchenne muscular dystrophy (DMD) is a progressive muscle disease caused by the loss of dystrophin, which results in inflammation, fibrosis, and the inhibition of myoblast differentiation in skeletal muscle. Catalpol, an iridoid glycoside, improves skeletal muscle function by enhancing my...

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Hlavní autoři: Dengqiu Xu, Lei Zhao, Jingwei Jiang, Sijia Li, Zeren Sun, Xiaofei Huang, Chunjie Li, Tao Wang, Lixin Sun, Xihua Li, Zhenzhou Jiang, Luyong Zhang
Médium: Artigo
Jazyk:Inglês
Vydáno: Wiley 2020-10-01
Edice:Journal of Cachexia, Sarcopenia and Muscle
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On-line přístup:https://doi.org/10.1002/jcsm.12581
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