Tlr4 Deficiency Protects against Cardiac Pressure Overload Induced Hyperinflammation.

Transverse aortic constriction provokes a pro-inflammatory reaction and results in cardiac hypertrophy. Endogenous ligands contribute to cardiac hypertrophy via toll-like receptor (TLR)-4 binding. A lack of TLR4 signaling diminishes hypertrophy and inflammation. Wild type mice undergoing aortic cons...

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Bibliografische Detailangaben
Hauptverfasser:	Heidi Ehrentraut, Stefan Felix Ehrentraut, Olaf Boehm, Sakina El Aissati, Fabian Foltz, Lina Goelz, David Goertz, Sied Kebir, Christina Weisheit, Michael Wolf, Rainer Meyer, Georg Baumgarten
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	Public Library of Science (PLoS) 2015-01-01
Schriftenreihe:	PLoS ONE
Online-Zugang:	https://doi.org/10.1371/journal.pone.0142921
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Tlr4 Deficiency Protects against Cardiac Pressure Overload Induced Hyperinflammation.

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