Propranolol reinstates mitochondrial dynamics and synaptic memory pathways through CaMKII/CREB–BDNF/ PKMζ cascades in an AD-like rat model

BackgroundAlzheimer’s disease (AD) is a major neurodegenerative disorder characterized by amyloid-β (Aβ) accumulation, neurofibrillary tangles, and progressive cognitive decline. Despite significant advances in understanding its pathophysiology, current therapeutic options provide limited symptomati...

Täydet tiedot

Bibliografiset tiedot

Päätekijät:	Pinky, Neha, Medha Kaushik, Prachi Tiwari, Mohamed El-Tanani, Syed Arman Rabbani, Suhel Parvez
Aineistotyyppi:	Artigo
Kieli:	Inglês
Julkaistu:	Frontiers Media S.A. 2026-05-01
Sarja:	Frontiers in Aging Neuroscience
Aiheet:	Alzheimer’s disease propranolol mitochondrial dynamics synaptic plasticity CaMKII/CREB signaling BDNF pathway
Linkit:	https://www.frontiersin.org/articles/10.3389/fnagi.2026.1729046/full
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