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Elevated Kallistatin Induces Myosteatosis and Exercise Intolerance by Antagonizing AdipoR1‐Mediated AMPK Signalling

ABSTRACT Background Pathological intramuscular lipid deposition (myosteatosis) and exercise intolerance are hallmarks of metabolic disorders, including diabetes and metabolic dysfunction‐associated steatotic liver disease (MASLD), yet their underlying mechanisms remain unclear. Our previous work has...

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Tallennettuna:
Päätekijät: Fuyan Hong, Zhenzhen Fang, Gang Shen, Yan Wang, Yunhua Li, Youbin Zhong, Junze Dai, Chengwei Zhang, Jing Zhang, Wencan Chen, Weiwei Qi, Xia Yang, Guoquan Gao, Ti Zhou
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Wiley 2026-04-01
Sarja:Journal of Cachexia, Sarcopenia and Muscle
Aiheet:
AdipoR1
AMPK
kallistatin
lipogenesis
mitochondrial biogenesis
myosteatosis
Linkit:https://doi.org/10.1002/jcsm.70261
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