Elevated sclerostin levels contribute to reduced bone mineral density in non-ambulatory stroke patients

Osteoporosis following stroke is a significant impediment to patient recovery. Decreased mechanical loading and locomotion following the onset of paralysis in stroke patients, especially those who are non-ambulatory, contributes greatly to bone loss. Sclerostin, a protein encoded by the SOST gene, a...

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Auteurs principaux:	Hye Kyoung Lee, Geneva Rose Notario, Sun Young Won, Jung Hwan Kim, Su Min Lee, Ha Seong Kim, Sung-Rae Cho
Format:	Artigo
Langue:	Inglês
Publié:	Elsevier 2025-06-01
Collection:	Bone Reports
Sujets:	Stroke Bone mineral density Sclerostin Ambulatory status Mechanical loading
Accès en ligne:	http://www.sciencedirect.com/science/article/pii/S2352187225000063
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