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IL-33 suppresses GSK-3β activation through an ST2-independent MyD88/TRAF6/RIP/PI3K/Akt pathway

Aims: The present study was conducted to explore the effect of interleukin-33 (IL-33) on glycogen synthase kinase-3β (GSK-3β) activation involving Tau phosphorylation, a critical causative factor for Alzheimer's disease (AD). Main methods: Experiments were performed using PC-12 cells. Target protein...

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Autore principale: Tomoyuki Nishizaki
Natura: Artigo
Lingua:Inglês
Pubblicazione: Elsevier 2018-11-01
Serie:Heliyon
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Accesso online:http://www.sciencedirect.com/science/article/pii/S2405844018363813
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