IL-33 suppresses GSK-3β activation through an ST2-independent MyD88/TRAF6/RIP/PI3K/Akt pathway
Aims: The present study was conducted to explore the effect of interleukin-33 (IL-33) on glycogen synthase kinase-3β (GSK-3β) activation involving Tau phosphorylation, a critical causative factor for Alzheimer's disease (AD). Main methods: Experiments were performed using PC-12 cells. Target protein...
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| Autore principale: | |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Elsevier
2018-11-01
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| Serie: | Heliyon |
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| Accesso online: | http://www.sciencedirect.com/science/article/pii/S2405844018363813 |
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