Dopamine enhances recovery after traumatic brain injury through ubiquitylation and autophagic degradation of RIPK1
Abstract Background Although the neurotransmitter dopamine (DA) plays a crucial pathophysiologic role after traumatic brain injury (TBI), its function and specific underlying mechanisms of action remain unclear. Methods Adult male mice underwent controlled cortical impact (CCI). We administered DA i...
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| Huvudupphov: | , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Utgiven: |
BMC
2024-02-01
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| Serie: | Cell Communication and Signaling |
| Ämnen: | |
| Länkar: | https://doi.org/10.1186/s12964-024-01515-y |
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