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The metabolic bone disease associated with the Hyp mutation is independent of osteoblastic HIF1α expression

Fibroblast growth factor-23 (FGF23) controls key responses to systemic phosphate increases through its phosphaturic actions on the kidney. In addition to stimulation by phosphate, FGF23 positively responds to iron deficiency anemia and hypoxia in rodent models and in humans. The disorder X-linked hy...

Бүрэн тодорхойлолт

-д хадгалсан:
Номзүйн дэлгэрэнгүй
Үндсэн зохиолчид: Julia M. Hum, Erica L. Clinkenbeard, Colin Ip, Taryn A. Cass, Matt Allen, Kenneth E. White
Формат: Artigo
Хэл сонгох:Inglês
Хэвлэсэн: Elsevier 2017-06-01
Цуврал:Bone Reports
Нөхцлүүд:
Онлайн хандалт:http://www.sciencedirect.com/science/article/pii/S2352187217300037
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