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TRIM26 facilitates PRV infection through NDP52-mediated autophagic degradation of MAVS

Abstract Pseudorabies virus (PRV) has evolved multiple strategies to evade host antiviral responses to benefit virus replication and establish persistent infection. Recently, tripartite motif 26 (TRIM26), a TRIM family protein, has been shown to be involved in a broad range of biological processes i...

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Auteurs principaux: Wu Chengyue, Wang Mengdong, Wang Xiaoquan, Chen Yeping, Li Hao, Sun Liumei, Ren Jianle, Zhang Zhendong
Format: Artigo
Langue:Inglês
Publié: BMC 2024-07-01
Collection:Veterinary Research
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Accès en ligne:https://doi.org/10.1186/s13567-024-01336-4
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