Redox-dependent activation of protein kinase G1α contributes to transient receptor potential cation channel subfamily V member 1-mediated acute nociceptive pain behavior

Background Sensory neurons relay the pain signals to the brain via the nociceptive system. Notably, reactive oxygen species (ROS) serve as signaling molecules in the somatosensory system; however, their contribution to sensing noxious stimuli remains poorly understood.Methods Herein, the role of pro...

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Autori principali:	Tim Berg, Katharina Metzner, Nabil Bahrami, Elena Wang, Maximilian Koch, Philip Eaton, Achim Schmidtko, Wiebke Kallenborn-Gerhardt
Natura:	Artigo
Lingua:	Inglês
Pubblicazione:	Taylor & Francis Group 2025-12-01
Serie:	Redox Report
Soggetti:	Pain nociception PKG1α reactive oxygen species TRPV1 capsaicin
Accesso online:	https://www.tandfonline.com/doi/10.1080/13510002.2025.2549954
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