ClC-3 mediates angiotensin II-induced endothelial dysfunction by inhibiting Akt-Hsp90-eNOS signaling pathway

BackgroundEndothelial dysfunction is a major driver for hypertension and diabetes. Chloride (Cl−) is the most abundant anion in vascular endothelial cells (ECs). However, the role of chloride channel or chloride/proton exchanger ClC-3 in the vascular endothelium and its relationship with endothelium...

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מידע ביבליוגרפי

Principais autores:	Lei Li, Yu-quan Lin, Yu-sheng Peng, Huan-hao Chen, Xiao-jun Wu, Guozheng Liang, Yan-hua Du
פורמט:	Artigo
שפה:	Inglês
יצא לאור:	Frontiers Media S.A. 2026-03-01
סדרה:	Frontiers in Pharmacology
נושאים:	angiotenin II ClC-3 endothelial dysfunction hypertension nitric oxide
גישה מקוונת:	https://www.frontiersin.org/articles/10.3389/fphar.2026.1795707/full
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