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SPARC activates p38γ signaling to promote PFKFB3 protein stabilization and contributes to keloid fibroblast glycolysis

Abstract Background Keloids are currently challenging to treat because they recur after resection which may affect patients’ quality of life. At present, no universal consensus on treatment regimen has been established. Thus, finding new molecular mechanisms underlying keloid formation is imminent....

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Principais autores: Yining Liu, Wei Zhang, Nan Lin, Zelei Yang, Yanxin Liu, Huaxia Chen
פורמט: Artigo
שפה:Inglês
יצא לאור: BMC 2024-10-01
סדרה:Inflammation and Regeneration
נושאים:
Keloid
Glycolysis
SPARC
P38γ
PFKFB3
גישה מקוונת:https://doi.org/10.1186/s41232-024-00357-y
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