Macrophage apoAI protects against dyslipidemia-induced dermatitis and atherosclerosis without affecting HDL[S]

Tissue cholesterol accumulation, macrophage infiltration, and inflammation are features of atherosclerosis and some forms of dermatitis. HDL and its main protein, apoAI, are acceptors of excess cholesterol from macrophages; this process inhibits tissue inflammation. Recent epidemiologic and clinical...

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Bibliografiska uppgifter

Huvudupphov:	Hagai Tavori, Yan Ru Su, Patricia G. Yancey, Ilaria Giunzioni, Ashley J. Wilhelm, John L. Blakemore, Manal Zabalawi, MacRae F. Linton, Mary G. Sorci-Thomas, Sergio Fazio
Materialtyp:	Artigo
Språk:	Inglês
Utgiven:	Elsevier 2015-03-01
Serie:	Journal of Lipid Research
Ämnen:	apolipoprotein AI high density lipoprotein gene therapy hematopoietic progenitor cells bone marrow transplant
Länkar:	http://www.sciencedirect.com/science/article/pii/S0022227520355917
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