Glabridin improves cardiac remodeling after myocardial infarction by activating PPARγ to regulate the ubiquitination and degradation of EGFR

Abstract Background Acute myocardial infarction leads to myocardial inflammation and necroptosis, and is a major cause of heart failure, cardiovascular dysfunction, and mortality. Adverse remodeling and myocardial fibrosis following myocardial infarction are key pathophysiological processes contribu...

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主要な著者:	Zixuan Li, Yiran Hu, Yu Jiang, Bingqi Fu, Tianxin Long, Hao Huang, Juwei Yang, Min Gu, Hongxia Niu, Wei Hua
フォーマット:	Artigo
言語:	Inglês
出版事項:	BMC 2026-04-01
シリーズ:	Biology Direct
主題:	Glabridin Myocardial infarction Fibrosis PPARγ EGFR PI3K/Akt pathway
オンライン･アクセス:	https://doi.org/10.1186/s13062-026-00809-0
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