Attenuating mitochondrial dysfunction and morphological disruption with PT320 delays dopamine degeneration in MitoPark mice

Abstract Background Mitochondria are essential organelles involved in cellular energy production. Changes in mitochondrial function can lead to dysfunction and cell death in aging and age-related disorders. Recent research suggests that mitochondrial dysfunction is closely linked to neurodegenerativ...

Täydet tiedot

Bibliografiset tiedot

Päätekijät:	Vicki Wang, Kuan-Yin Tseng, Tung-Tai Kuo, Eagle Yi-Kung Huang, Kuo-Lun Lan, Zi-Rong Chen, Kuo-Hsing Ma, Nigel H. Greig, Jin Jung, Ho-II Choi, Lars Olson, Barry J. Hoffer, Yuan-Hao Chen
Aineistotyyppi:	Artigo
Kieli:	Inglês
Julkaistu:	BMC 2024-04-01
Sarja:	Journal of Biomedical Science
Aiheet:	Glucagon-like peptide-1 receptor agonist MitoPark mouse Parkinson’s disease Exenatide Mitochondria PT320
Linkit:	https://doi.org/10.1186/s12929-024-01025-6
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