Cardiomyocyte NOX4 regulates resident macrophage-mediated inflammation and diastolic dysfunction in stress cardiomyopathy

In acute sympathetic stress, catecholamine overload can lead to stress cardiomyopathy. We tested the hypothesis that cardiomyocyte NOX4 (NADPH oxidase 4)-dependent mitochondrial oxidative stress mediates inflammation and diastolic dysfunction in stress cardiomyopathy. Isoproterenol (ISO; 5 mg/kg) in...

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Λεπτομέρειες βιβλιογραφικής εγγραφής

Κύριοι συγγραφείς:	Aleksandr E. Vendrov, Han Xiao, Andrey Lozhkin, Takayuki Hayami, Guomin Hu, Matthew J. Brody, Junichi Sadoshima, You-Yi Zhang, Marschall S. Runge, Nageswara R. Madamanchi
Μορφή:	Artigo
Γλώσσα:	Inglês
Έκδοση:	Elsevier 2023-11-01
Σειρά:	Redox Biology
Θέματα:	Cardiomyopathy Cardiomyocyte mitochondria Resident macrophages Cardiac fibroblasts NADPH oxidase 4 Diastolic dysfunction
Διαθέσιμο Online:	http://www.sciencedirect.com/science/article/pii/S2213231723003385
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