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Inhibition of RIPK1/RIPK3-MLKL inflammatory signaling pathway activation attenuates preterm birth

Abstract Preterm birth (PTB) is a principal contributor to neonatal morbidity, wherein inflammation and dysregulated cell death pathways are implicated as key drivers in its pathogenesis. However, the role of the RIPK1/RIPK3-MLKL signaling axis, a critical regulator of necroptosis and inflammatory r...

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Principais autores: Xuexiang Bing, Yongqing Wang, Jiacui Zheng, Guodong Gao, Jinxiao Jiang, Lanlan Liu, Xue Zhang
Format: Artigo
Jezik:Inglês
Izdano: Nature Publishing Group 2026-04-01
Serija:Cell Death Discovery
Online dostop:https://doi.org/10.1038/s41420-026-03093-z
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