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Zebrafish nampt-a mutants are viable despite perturbed primitive hematopoiesis

Abstract Background Nicotinamide phosphoribosyltransferase (Nampt) is required for recycling NAD+ in numerous cellular contexts. Morpholino-based knockdown of zebrafish nampt-a has been shown to cause abnormal development and defective hematopoiesis concomitant with decreased NAD+ levels. However, s...

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Auteurs principaux: Autumn Penecilla Pomreinke, Patrick Müller
Format: Artigo
Langue:Inglês
Publié: BMC 2024-04-01
Collection:Hereditas
Accès en ligne:https://doi.org/10.1186/s41065-024-00318-y
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