Zebrafish nampt-a mutants are viable despite perturbed primitive hematopoiesis
Abstract Background Nicotinamide phosphoribosyltransferase (Nampt) is required for recycling NAD+ in numerous cellular contexts. Morpholino-based knockdown of zebrafish nampt-a has been shown to cause abnormal development and defective hematopoiesis concomitant with decreased NAD+ levels. However, s...
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| Hlavní autoři: | , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
BMC
2024-04-01
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| Edice: | Hereditas |
| On-line přístup: | https://doi.org/10.1186/s41065-024-00318-y |
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