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SLC38A1 drives malignant progression of hepatocellular carcinoma by activating PI3K/AKT signaling via enhanced glutamine uptake

Abstract Objective To investigate the mechanism by which SLC38A1 promotes hepatocellular carcinoma (HCC) progression through glutamine transport-mediated activation of the PI3K/AKT signaling pathway. Methods Bioinformatics analysis was employed to assess the correlation between SLC38A1 expression...

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Príomhchruthaitheoirí: YANG Yuanyuan, ZHANG Ping, HU Peng
Formáid: Artigo
Teanga:Chinês
Foilsithe / Cruthaithe: Editorial Office of Journal of Army Medical University 2025-12-01
Sraith:陆军军医大学学报
Ábhair:
Rochtain ar líne:https://aammt.tmmu.edu.cn/html/202508018.html
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