Antioxidant dipeptide, cyclo (Phe-Phe), protects against bone dysplasia by suppression of phospholipid peroxidation

Prenatal exposure to dexamethasone (Dex) has been identified as a significant factor that impairs longitudinal skeletal growth in fetuses, resulting in notable deficiencies in bone development. However, effective treatment options are currently limited due to the lack of understanding of the underly...

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Hlavní autoři:	Qiong-Yi Zhang, Rong-Ping Huang, Fang Wu, Hui-Er Ye, Chang-Yu Yan, Hai-Biao Gong, Cai-Ling Xu, Xi-You Li, Shao-Cong Han, Xiao-Min Li, Lei Liang, Hiroshi Kurihara, Yi-Fang Li, Rong-Rong He
Médium:	Artigo
Jazyk:	Inglês
Vydáno:	KeAi Communications Co., Ltd. 2025-06-01
Edice:	Cell Investigation
Témata:	Protein hydrolysate Cyclo (Phe-Phe) Glutathione peroxidase 4 Bone development defects Phospholipid peroxidation Ferroptosis
On-line přístup:	http://www.sciencedirect.com/science/article/pii/S3050538024000036
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