Luseogliflozin restores GIP responsiveness in diabetic male mice with preserved β‐cell function
Abstract Impaired glucose‐dependent insulinotropic polypeptide (GIP)‐induced insulin secretion is a characteristic feature of type 2 diabetes, whereas glucagon‐like peptide‐1 (GLP‐1) responsiveness is relatively preserved. Our previous study proposed that chronic β‐cell depolarization alters G prote...
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| Huvudupphov: | , , , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Utgiven: |
Wiley
2026-07-01
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| Serie: | Physiological Reports |
| Ämnen: | |
| Länkar: | https://doi.org/10.14814/phy2.71016 |
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