REST contributes to renal fibrosis through inducing mitochondrial energy metabolism imbalance in tubular epithelial cells

Abstract Background Renal fibrosis represents the final common pathological manifestation of chronic kidney disease (CKD), yet the underlying mechanism remains elusive, and there is still a lack of effective targeted therapeutic strategy. Although previous research indicated that repressor element 1...

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Λεπτομέρειες βιβλιογραφικής εγγραφής

Κύριοι συγγραφείς:	Yingxian Yang, Shuiqin Gong, Chun Zhou, Wang Xin, Shaozong Qin, Mengying Yao, Qigang Lan, Wenhao Liao, Jinghong Zhao, Yinghui Huang
Μορφή:	Artigo
Γλώσσα:	Inglês
Έκδοση:	BMC 2025-04-01
Σειρά:	Cell Communication and Signaling
Θέματα:	REST Renal fibrosis Fatty acid oxidation OGDHL
Διαθέσιμο Online:	https://doi.org/10.1186/s12964-025-02166-3
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