Presenilin1 exerts antiproliferative effects by repressing the Wnt/β-catenin pathway in glioblastoma
Abstract Background Glioblastoma and Alzheimer’s disease (AD) are the most common and devastating diseases in the central nervous system. The dysfunction of Presenilin1 is the main reason for AD pathogenesis. However, the molecular function of Presenilin1 and its relative mechanism in glioblastoma r...
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| Auteurs principaux: | , , , , , , |
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| Format: | Artigo |
| Langue: | Inglês |
| Publié: |
BMC
2020-02-01
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| Collection: | Cell Communication and Signaling |
| Sujets: | |
| Accès en ligne: | https://doi.org/10.1186/s12964-019-0501-9 |
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