Just like the rest of evolution in Mother Nature, the evolution of cancers may be driven by natural selection, and not by haphazard mutations

Por Zhang, Ju, Lou, Xiaomin, Zellmer, Lucas, Liu, Siqi, Xu, Ningzhi, Liao, D. Joshua
Publicado no Oncoscience (2014)

Hypothesis: Artifacts, Including Spurious Chimeric RNAs with a Short Homologous Sequence, Caused by Consecutive Reverse Transcriptions and Endogenous Random Primers

Por Peng, Zhiyu, Yuan, Chengfu, Zellmer, Lucas, Liu, Siqi, Xu, Ningzhi, Liao, D. Joshua
Publicado no J Cancer (2015)

Protecting the normal in order to better kill the cancer

Por Liu, Bingya, Ezeogu, Lewis, Zellmer, Lucas, Yu, Baofa, Xu, Ningzhi, Joshua Liao, Dezhong
Publicado no Cancer Med (2015)

Learning about the Importance of Mutation Prevention from Curable Cancers and Benign Tumors

Por Wang, Gangshi, Chen, Lichan, Yu, Baofa, Zellmer, Lucas, Xu, Ningzhi, Liao, D. Joshua
Publicado no J Cancer (2016)

It Is Imperative to Establish a Pellucid Definition of Chimeric RNA and to Clear Up a Lot of Confusion in the Relevant Research

Por Yuan, Chengfu, Han, Yaping, Zellmer, Lucas, Yang, Wenxiu, Guan, Zhizhong, Yu, Wenfeng, Huang, Hai, Liao, D. Joshua
Publicado no Int J Mol Sci (2017)

Probably less than one-tenth of the genes produce only the wild type protein without at least one additional protein isoform in some human cancer cell lines

Por Yan, Rui, Zhang, Ju, Zellmer, Lucas, Chen, Lichan, Wu, Di, Liu, Siqi, Xu, Ningzhi, Liao, Joshua D.
Publicado no Oncotarget (2017)