Serum Levels of Toxic AGEs (TAGE) May Be a Promising Novel Biomarker for the Onset/Progression of Lifestyle-Related Diseases

par Takeuchi, Masayoshi
Publié dans Diagnostics (Basel) (2016)

Toxic AGEs (TAGE) theory: a new concept for preventing the development of diseases related to lifestyle

par Takeuchi, Masayoshi
Publié dans Diabetol Metab Syndr (2020)

Glycer-AGEs-RAGE signaling enhances the angiogenic potential of hepatocellular carcinoma by upregulating VEGF expression

par Takino, Junichi, Yamagishi, Shoichi, Takeuchi, Masayoshi
Publié 2012

Impact of intracellular toxic advanced glycation end-products (TAGE) on murine myoblast cell death

par Takata, Takanobu, Sakasai-Sakai, Akiko, Takeuchi, Masayoshi
Publié dans Diabetol Metab Syndr (2020)

Intracellular Toxic Advanced Glycation End-Products Promote the Production of Reactive Oxygen Species in HepG2 Cells

par Sakasai-Sakai, Akiko, Takata, Takanobu, Takeuchi, Masayoshi
Publié dans Int J Mol Sci (2020)

Cancer Malignancy Is Enhanced by Glyceraldehyde-Derived Advanced Glycation End-Products

par Takino, Jun-Ichi, Yamagishi, Sho-Ichi, Takeuchi, Masayoshi
Publié 2010

Involvement of TAGE-RAGE System in the Pathogenesis of Diabetic Retinopathy

par Takeuchi, Masayoshi, Takino, Jun-ichi, Yamagishi, Sho-ichi
Publié 2010

Insulin resistance is an independent correlate of high serum levels of advanced glycation end products (AGEs) and low testosterone in non-diabetic men

par Tahara, Nobuhiro, Imaizumi, Tsutomu, Takeuchi, Masayoshi, Yamagishi, Sho-ichi
Publié 2010

In vitro identification of nonalcoholic fatty liver disease-related protein hnRNPM

par Takino, Jun-ichi, Nagamine, Kentaro, Takeuchi, Masayoshi, Hori, Takamitsu
Publié dans World J Gastroenterol (2015)

Generation of glyceraldehyde-derived advanced glycation end-products in pancreatic cancer cells and the potential of tumor promotion

par Takata, Takanobu, Ueda, Tadashi, Sakasai-Sakai, Akiko, Takeuchi, Masayoshi
Publié dans World J Gastroenterol (2017)

Intracellular toxic advanced glycation end-products in cardiomyocytes may cause cardiovascular disease

par Takata, Takanobu, Sakasai-Sakai, Akiko, Ueda, Tadashi, Takeuchi, Masayoshi
Publié dans Sci Rep (2019)

Impact of intracellular glyceraldehyde-derived advanced glycation end-products on human hepatocyte cell death

par Sakasai-Sakai, Akiko, Takata, Takanobu, Takino, Jun-ichi, Takeuchi, Masayoshi
Publié dans Sci Rep (2017)

The Relevance of Toxic AGEs (TAGE) Cytotoxicity to NASH Pathogenesis: A Mini-Review

par Sakasai-Sakai, Akiko, Takata, Takanobu, Takino, Jun-ichi, Takeuchi, Masayoshi
Publié dans Nutrients (2019)

Evidence for Toxic Advanced Glycation End-Products Generated in the Normal Rat Liver

par Takata, Takanobu, Sakasai-Sakai, Akiko, Takino, Jun-ichi, Takeuchi, Masayoshi
Publié dans Nutrients (2019)

The Effect of Glyceraldehyde-Derived Advanced Glycation End Products on β-Tubulin-Inhibited Neurite Outgrowth in SH-SY5Y Human Neuroblastoma Cells

par Nasu, Ryuto, Furukawa, Ayako, Suzuki, Keita, Takeuchi, Masayoshi, Koriyama, Yoshiki
Publié dans Nutrients (2020)

Glyceraldehyde caused Alzheimer’s disease-like alterations in diagnostic marker levels in SH-SY5Y human neuroblastoma cells

par Koriyama, Yoshiki, Furukawa, Ayako, Muramatsu, Michiru, Takino, Jun-ichi, Takeuchi, Masayoshi
Publié dans Sci Rep (2015)

Contribution of the toxic advanced glycation end-products-receptor axis in nonalcoholic steatohepatitis-related hepatocellular carcinoma

par Takino, Jun-ichi, Nagamine, Kentaro, Hori, Takamitsu, Sakasai-Sakai, Akiko, Takeuchi, Masayoshi
Publié dans World J Hepatol (2015)

Toxic AGE (TAGE) Theory for the Pathophysiology of the Onset/Progression of NAFLD and ALD

par Takeuchi, Masayoshi, Takino, Jun-ichi, Sakasai-Sakai, Akiko, Takata, Takanobu, Tsutsumi, Mikihiro
Publié dans Nutrients (2017)

Glyceraldehyde-Derived Advanced Glycation End Products Accumulate Faster Than N(ε)-(Carboxymethyl) Lysine

par Yokota, Mami, Sekita, Marie, Okano, Yuri, Masaki, Hitoshi, Takeuchi, Masayoshi, Tokudome, Yoshihiro
Publié dans Ann Dermatol (2017)

Beraprost sodium, a prostaglandin I2 analogue, protects against advanced gycation end products-induced injury in cultured retinal pericytes.

par Yamagishi, Sho-ichi, Amano, Shinjiro, Inagaki, Yosuke, Okamoto, Tamami, Takeuchi, Masayoshi, Makita, Zenji
Publié 2002