The neuronal cell cycle as a mechanism of pathogenesis in Alzheimer's disease

بواسطة Currais, Antonio, Hortobágyi, Tibor, Soriano, Salvador
منشور في 2009

Moving beyond anti-amyloid therapy for the prevention and treatment of Alzheimer’s disease

بواسطة Castello, Michael A, Jeppson, John David, Soriano, Salvador
الحاوية / القاعدة BMC Neurol (2014)

Perturbed Neurogenesis in the Adult Hippocampus Associated with Presenilin-1 A246E Mutation

بواسطة Chevallier, Nathalie L., Soriano, Salvador, Kang, David E., Masliah, Eliezer, Hu, Gloria, Koo, Edward H.
منشور في 2005

Presenilin 1 Negatively Regulates β-Catenin/T Cell Factor/Lymphoid Enhancer Factor-1 Signaling Independently of β-Amyloid Precursor Protein and Notch Processing

بواسطة Soriano, Salvador, Kang, David E., Fu, Maofu, Pestell, Richard, Chevallier, Nathalie, Zheng, Hui, Koo, Edward H.
منشور في 2001

Stability and Bioactivity of Chitosan as a Transfection Agent in Primary Human Cell Cultures: A Case for Chitosan-only Controls: Chitosan Modulates CD59 Expression

بواسطة Cupino, Tanya L., Watson, Billy A., Cupino, Alan C., Oda, Keiji, Ghamsary, Mark G., Soriano, Salvador, Kirsch, Wolff M.
الحاوية / القاعدة Carbohydr Polym (2017)

Loss of amyloid precursor protein exacerbates early inflammation in Niemann-Pick disease type C

بواسطة Shin, Samuel D., Shin, Alexandra, Mayagoitia, Karina, Siebold, Lorraine, Rubini, Marsilio, Wilson, Christopher G., Bellinger, Denise L., Soriano, Salvador
الحاوية / القاعدة J Neuroinflammation (2019)

Loss of presenilin 1 is associated with enhanced β-catenin signaling and skin tumorigenesis

بواسطة Xia, Xuefeng, Qian, Su, Soriano, Salvador, Wu, Ying, Fletcher, Anthony M., Wang, Xiao-Jing, Koo, Edward H., Wu, Xiangwei, Zheng, Hui
منشور في 2001

Presenilin 1 Facilitates the Constitutive Turnover of β-Catenin: Differential Activity of Alzheimer’s Disease–Linked PS1 Mutants in the β-Catenin–Signaling Pathway

بواسطة Kang, David E., Soriano, Salvador, Frosch, Matthew P., Collins, Tucker, Naruse, Satoshi, Sisodia, Sangram S., Leibowitz, Gil, Levine, Fred, Koo, Edward H.
الحاوية / القاعدة J Neurosci (1999)

The aspartate-257 of presenilin 1 is indispensable for mouse development and production of β-amyloid peptides through β-catenin-independent mechanisms

بواسطة Xia, Xuefeng, Wang, Pei, Sun, Xiaoyan, Soriano, Salvador, Shum, Wan-Kyng, Yamaguchi, Haruyasu, Trumbauer, Myrna E., Takashima, Akihiko, Koo, Edward H., Zheng, Hui
منشور في 2002

Cellular hormetic response to 27-hydroxycholesterol promotes neuroprotection through AICD induction of MAST4 abundance and kinase activity

بواسطة Gongol, Brendan, Marin, Traci L., Jeppson, John D., Mayagoitia, Karina, Shin, Samuel, Sanchez, Nicholas, Kirsch, Wolff M., Vinters, Harry V., Wilson, Christopher G., Ghribi, Othman, Soriano, Salvador
الحاوية / القاعدة Sci Rep (2017)

Interaction between Oligomers of Stefin B and Amyloid-β in Vitro and in Cells

بواسطة Škerget, Katja, Taler-Verčič, Ajda, Bavdek, Andrej, Hodnik, Vesna, Čeru, Slavko, Tušek-Žnidarič, Magda, Kumm, Tiina, Pitsi, Didier, Pompe-Novak, Maruša, Palumaa, Peep, Soriano, Salvador, Kopitar-Jerala, Nataša, Turk, Vito, Anderluh, Gregor, Žerovnik, Eva
منشور في 2010