Low levels of vitronectin and clusterin in acute meningococcal disease are closely associated with formation of the terminal-complement complex and the vitronectin-thrombin-antithr...

Por Høgåsen, K, Mollnes, T E, Brandtzaeg, P
Publicado em 1994

Extensive complement activation in hereditary porcine membranoproliferative glomerulonephritis type II (porcine dense deposit disease).

Por Jansen, J. H., Høgåsen, K., Mollnes, T. E.
Publicado em 1993

Hereditary porcine membranoproliferative glomerulonephritis type II is caused by factor H deficiency.

Por Høgåsen, K, Jansen, J H, Mollnes, T E, Hovdenes, J, Harboe, M
Publicado em 1995

Acquired C3 deficiency in patients with alcoholic cirrhosis predisposes to infection and increased mortality.

Por Homann, C, Varming, K, Høgåsen, K, Mollnes, T E, Graudal, N, Thomsen, A C, Garred, P
Publicado em 1997

Complement activation and bioincompatibility. The terminal complement complex for evaluation and surface modification with heparin for improvement of biomaterials.

Por Mollnes, T E, Videm, V, Riesenfeld, J, Garred, P, Svennevig, J L, Fosse, E, Hogasen, K, Harboe, M
Publicado em 1991

Complement activation in septic baboons detected by neoepitope-specific assays for C3b/iC3b/C3c, C5a and the terminal C5b-9 complement complex (TCC).

Por Mollnes, T E, Redl, H, Høgåsen, K, Bengtsson, A, Garred, P, Speilberg, L, Lea, T, Oppermann, M, Götze, O, Schlag, G
Publicado em 1993