A1152D mutation of the Na(+) channel causes paramyotonia congenita and emphasizes the role of DIII/S4–S5 linker in fast inactivation

по Bouhours, Magali, Luce, Sandrine, Sternberg, Damien, Willer, Jean Claude, Fontaine, Bertrand, Tabti, Nacira
Опубликовано 2005

Functional characterization and cold sensitivity of T1313A, a new mutation of the skeletal muscle sodium channel causing paramyotonia congenita in humans

по Bouhours, Magali, Sternberg, Damien, Davoine, Claire-Sophie, Xavier, Ferrer, Willer, Jean Claude, Fontaine, Bertrand, Tabti, Nacira
Опубликовано 2004

A Co-operative Regulation of Neuronal Excitability by UNC-7 Innexin and NCA/NALCN Leak Channel

по Bouhours, Magali, Po, Michelle D, Gao, Shangbang, Hung, Wesley, Li, Hang, Georgiou, John, Roder, John C, Zhen, Mei
Опубликовано 2011

A Putative Cation Channel, NCA-1, and a Novel Protein, UNC-80, Transmit Neuronal Activity in C. elegans

по Yeh, Edward, Ng, Sharon, Zhang, Mi, Bouhours, Magali, Wang, Ying, Wang, Min, Hung, Wesley, Aoyagi, Kyota, Melnik-Martinez, Katya, Li, Michelle, Liu, Fang, Schafer, William R, Zhen, Mei
Опубликовано 2008

ITSN-1 Controls Vesicle Recycling at the Neuromuscular Junction and Functions in Parallel with DAB-1

по Wang, Wei, Bouhours, Magali, Gracheva, Elena O., Liao, Edward H., Xu, Keli, Sengar, Ameet S., Xin, Xiaofeng, Roder, John, Boone, Charles, Richmond, Janet E., Zhen, Mei, Egan, Sean E.
Опубликовано 2008