A1152D mutation of the Na(+) channel causes paramyotonia congenita and emphasizes the role of DIII/S4–S5 linker in fast inactivation

Por Bouhours, Magali, Luce, Sandrine, Sternberg, Damien, Willer, Jean Claude, Fontaine, Bertrand, Tabti, Nacira
Publicado em 2005

Functional characterization and cold sensitivity of T1313A, a new mutation of the skeletal muscle sodium channel causing paramyotonia congenita in humans

Por Bouhours, Magali, Sternberg, Damien, Davoine, Claire-Sophie, Xavier, Ferrer, Willer, Jean Claude, Fontaine, Bertrand, Tabti, Nacira
Publicado em 2004

A Co-operative Regulation of Neuronal Excitability by UNC-7 Innexin and NCA/NALCN Leak Channel

Por Bouhours, Magali, Po, Michelle D, Gao, Shangbang, Hung, Wesley, Li, Hang, Georgiou, John, Roder, John C, Zhen, Mei
Publicado em 2011

A Putative Cation Channel, NCA-1, and a Novel Protein, UNC-80, Transmit Neuronal Activity in C. elegans

Por Yeh, Edward, Ng, Sharon, Zhang, Mi, Bouhours, Magali, Wang, Ying, Wang, Min, Hung, Wesley, Aoyagi, Kyota, Melnik-Martinez, Katya, Li, Michelle, Liu, Fang, Schafer, William R, Zhen, Mei
Publicado em 2008

ITSN-1 Controls Vesicle Recycling at the Neuromuscular Junction and Functions in Parallel with DAB-1

Por Wang, Wei, Bouhours, Magali, Gracheva, Elena O., Liao, Edward H., Xu, Keli, Sengar, Ameet S., Xin, Xiaofeng, Roder, John, Boone, Charles, Richmond, Janet E., Zhen, Mei, Egan, Sean E.
Publicado em 2008