Nrp-1 Mediated Plasmatic Ago2 Binding miR-21a-3p Internalization: A Novel Mechanism for miR-21a-3p Accumulation in Renal Tubular Epithelial Cells during Sepsis

The mechanism underlying sepsis-associated acute kidney injury (SAKI), which is an independent risk factor for sepsis-associated death, is unclear. A previous study indicates that during sepsis miR-21a-3p accumulates in renal tubular epithelial cells (TECs) as the mediator of inflammation and mediat...

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Bibliografiska uppgifter
I publikationen:Biomed Res Int
Huvudupphovsmän: Zou, Zhiqiang, Lin, Qin, Yang, Huobao, Liu, Zhongwei, Zheng, Shixiang
Materialtyp: Artigo
Språk:Inglês
Publicerad: Hindawi 2020
Ämnen:
Research Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC7453242/
https://ncbi.nlm.nih.gov/pubmed/32923478
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1155/2020/2370253
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