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Nrp-1 Mediated Plasmatic Ago2 Binding miR-21a-3p Internalization: A Novel Mechanism for miR-21a-3p Accumulation in Renal Tubular Epithelial Cells during Sepsis
The mechanism underlying sepsis-associated acute kidney injury (SAKI), which is an independent risk factor for sepsis-associated death, is unclear. A previous study indicates that during sepsis miR-21a-3p accumulates in renal tubular epithelial cells (TECs) as the mediator of inflammation and mediat...
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出版年: | Biomed Res Int |
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主要な著者: | , , , , |
フォーマット: | Artigo |
言語: | Inglês |
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Hindawi
2020
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主題: | |
オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7453242/ https://ncbi.nlm.nih.gov/pubmed/32923478 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1155/2020/2370253 |
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