Role of MKP-5-p38/MAPK pathway in Clopidogrel-induced gastric mucosal epithelial cells apoptosis and tight junction dysfunction

Bleeding and delayed healing of gastric ulcer are well-recognized in patients following Clopidorgrel treatment. Our previous studies have shown that endoplasmic reticulum stress (ER) is involved in Clopidogrel-induced gastric mucosal damage through activating p38 mitogen-activated protein kinases (M...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:Am J Transl Res
Päätekijät: Wu, Hailu, Ming, Wei, Tan, Jiacheng, Lu, Qin, Mulmi Shrestha, Sachin, Li, Nan, Wu, Guoqiu, Zhang, Zhenyu, Shi, Ruihua
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: e-Century Publishing Corporation 2020
Aiheet:
Original Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC7269986/
https://ncbi.nlm.nih.gov/pubmed/32509173
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