Rassadi, S., Krishnaswamy, A., Pié, B., McConnell, R., Jacob, M. H., & Cooper, E. (2005). A Null Mutation for the α3 Nicotinic Acetylcholine (ACh) Receptor Gene Abolishes Fast Synaptic Activity in Sympathetic Ganglia and Reveals That ACh Output from Developing Preganglionic Terminals Is Regulated in an Activity-Dependent Retrograde Manner. J Neurosci.
Chicago Style CitationRassadi, Siamak, Arjun Krishnaswamy, Brigitte Pié, Russell McConnell, Michele H. Jacob, i Ellis Cooper. "A Null Mutation for the α3 Nicotinic Acetylcholine (ACh) Receptor Gene Abolishes Fast Synaptic Activity in Sympathetic Ganglia and Reveals That ACh Output From Developing Preganglionic Terminals Is Regulated in an Activity-Dependent Retrograde Manner." J Neurosci 2005.
Cita MLARassadi, Siamak, et al. "A Null Mutation for the α3 Nicotinic Acetylcholine (ACh) Receptor Gene Abolishes Fast Synaptic Activity in Sympathetic Ganglia and Reveals That ACh Output From Developing Preganglionic Terminals Is Regulated in an Activity-Dependent Retrograde Manner." J Neurosci 2005.