טוען...
Axoplasm enriched in a protein mobilized by nerve injury induces memory- like alterations in Aplysia neurons
Axon regeneration after injury and long-term alterations associated with learning both require protein synthesis in the neuronal cell body, but the signals that initiate these changes are largely unknown. Direct evidence that axonal injury activates molecular signals in the axon was obtained by inje...
שמור ב:
| הוצא לאור ב: | J Neurosci |
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| Main Authors: | , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Society for Neuroscience
1995
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6578185/ https://ncbi.nlm.nih.gov/pubmed/7538559 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.15-05-03440.1995 |
| תגים: |
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