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Chronic synaptic insulin resistance after traumatic brain injury abolishes insulin protection from amyloid beta and tau oligomer-induced synaptic dysfunction

Traumatic brain injury (TBI) is a risk factor for Alzheimer’s disease (AD), although the mechanisms contributing to this increased risk are unknown. Insulin resistance is an additional risk factor for AD whereby decreased insulin signaling increases synaptic sensitivity to amyloid beta (Aβ) and tau....

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Pubblicato in:Sci Rep
Autori principali: Franklin, Whitney, Krishnan, Balaji, Taglialatela, Giulio
Natura: Artigo
Lingua:Inglês
Pubblicazione: Nature Publishing Group UK 2019
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6546708/
https://ncbi.nlm.nih.gov/pubmed/31160730
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-019-44635-z
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