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Chromatin stability as a target for cancer treatment
In this essay, I propose that DNA-binding anti-cancer drugs work more via chromatin disruption than DNA damage. Success of long-awaited drugs targeting cancer-specific drivers is limited by the heterogeneity of tumors. Therefore, chemotherapy acting via universal targets (e.g., DNA) is still the mai...
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| Publicado no: | Bioessays |
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| Autor principal: | |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6522245/ https://ncbi.nlm.nih.gov/pubmed/30566250 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/bies.201800141 |
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