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STAT6 degradation and ubiquitylated TRIML2 are essential for activation of human oncogenic herpesvirus

Aberrations in STAT6-mediated signaling are linked to the development of multiple cancer types. Increasing evidence has shown that activation of human oncogenic herpesvirus lytic replication is crucial for viral tumorigenesis. However, the role of STAT6 in herpesvirus lytic replication remains elusi...

Täydet tiedot

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Bibliografiset tiedot
Julkaisussa:PLoS Pathog
Päätekijät: Gu, Feng, Wang, Chong, Wei, Fang, Wang, Yuyan, Zhu, Qing, Ding, Ling, Xu, Wenjia, Zhu, Caixia, Cai, Cankun, Qian, Zhikang, Yuan, Zhenghong, Robertson, Erle, Cai, Qiliang
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Public Library of Science 2018
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6287816/
https://ncbi.nlm.nih.gov/pubmed/30532138
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.ppat.1007416
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