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Autophagy is increased following either pharmacological or genetic silencing of mGluR5 signaling in Alzheimer’s disease mouse models

Alzheimer’s disease (AD) is characterized by neurotoxicity mediated by the accumulation of beta amyloid (Aβ) oligomers, causing neuronal loss and progressive cognitive decline. Genetic deletion or chronic pharmacological inhibition of mGluR5 by the negative allosteric modulator CTEP, rescues cogniti...

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Detalhes bibliográficos
Publicado no:Mol Brain
Main Authors: Abd-Elrahman, Khaled S., Hamilton, Alison, Vasefi, Maryam, Ferguson, Stephen S. G.
Formato: Artigo
Idioma:Inglês
Publicado em: BioMed Central 2018
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5892040/
https://ncbi.nlm.nih.gov/pubmed/29631635
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13041-018-0364-9
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