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Gα(i)-mediated TRPC4 activation by polycystin-1 contributes to endothelial function via STAT1 activation
Hypertension and aneurysm are frequently associated with autosomal dominant polycystic kidney disease (ADPKD) caused by polycystin-1 (PC1) mutations, which is closely related to endothelial dysfunction. PC1 is an atypical G-protein-coupled receptor that activates G-proteins by self-cleavage; current...
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| Publicado no: | Sci Rep |
|---|---|
| Main Authors: | , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group UK
2018
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5823873/ https://ncbi.nlm.nih.gov/pubmed/29472562 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-018-21873-1 |
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