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Targeting trisomic treatments: optimizing Dyrk1a inhibition to improve Down syndrome deficits
Overexpression of Dual‐specificity tyrosine‐phosphorylated regulated kinase 1A (DYRK1A), located on human chromosome 21, may alter molecular processes linked to developmental deficits in Down syndrome (DS). Trisomic DYRK1A is a rational therapeutic target, and although reductions in Dyrk1a genetic d...
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| Publicado no: | Mol Genet Genomic Med |
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| Main Authors: | , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
John Wiley and Sons Inc.
2017
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5606891/ https://ncbi.nlm.nih.gov/pubmed/28944229 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/mgg3.334 |
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