Shackman, A. J., Fox, A. S., Oler, J. A., Shelton, S. E., Oakes, T. R., Davidson, R. J., & Kalin, N. H. (2016). Heightened extended amygdala metabolism following threat characterizes the early phenotypic risk to develop anxiety-related psychopathology. Mol Psychiatry.
Citación estilo ChicagoShackman, Alexander J., Andrew S. Fox, Jonathan A. Oler, Steven E. Shelton, Terrence R. Oakes, Richard J. Davidson, and Ned H. Kalin. "Heightened Extended Amygdala Metabolism Following Threat Characterizes the Early Phenotypic Risk to Develop Anxiety-related Psychopathology." Mol Psychiatry 2016.
Cita MLAShackman, Alexander J., et al. "Heightened Extended Amygdala Metabolism Following Threat Characterizes the Early Phenotypic Risk to Develop Anxiety-related Psychopathology." Mol Psychiatry 2016.